PEPTIVIAMOTS-C10 mgVIAL · RESEARCH USE ONLY1 × 10 mg
2 VIALS · 2 DISTINCT MATERIALS
Explore the chemistry of cellular energy.
Cellular Energy Stack
NAD+ and MOTS-c connect a fundamental coenzyme with a mitochondrial-derived peptide. Explore redox reactions, metabolic sensing and the cellular response to stress through two distinct biological roles.
CURATED INDIVIDUAL ITEMS · TEST / DEMOAT A GLANCE
Inside the stack
NAD+
1 × 500 mg single vial · $99.99 CAD each
$99.99 CAD
NAD+ is a coenzyme at the centre of cellular redox chemistry. Its role in electron transfer makes it a foundational material for exploring energy metabolism and enzyme activity.
Redox reactions — Nicotinamide adenine dinucleotide participates in electron-transfer reactions.
Energy metabolism — NAD-dependent reactions connect nutrient processing with cellular energy chemistry.
Coenzyme research — A non-peptide identity supports biochemical questions distinct from receptor-targeting peptides.
MOTS-c is a mitochondrial-derived peptide studied for cellular metabolic adaptation. Cell and mouse research explores AMPK-related signalling and how cells respond to metabolic stress.
Metabolic sensing — AMPK-related pathways connect cellular energy status with adaptive responses.
Stress signalling — Experiments investigate stress-responsive movement into the nucleus and changes in gene expression.
Mitochondrial communication — A peptide encoded within mitochondrial DNA offers a distinct perspective on communication within cells.
NAD+ carries electrons in redox reactions that underpin energy metabolism. MOTS-c is studied for AMPK-related signalling and stress-responsive nuclear activity. One is a coenzyme, the other a peptide signal; their individual roles make the research connection useful without establishing a combined human energy benefit.
Explore the evidence & study context
NAD+
Coenzyme (not a peptide) · redox and energy-metabolism research
No reviewed study findings supplied for this identity. Parent-peptide evidence is not a substitute.
MOTS-C
Cells / animal
The 2015 study linked MOTS-c to AMPK-related cellular signalling and reported altered insulin-resistance outcomes in mouse models.
Cells and mice, not a human efficacy trial or an energy/weight-loss claim for PEPTIVIA material.
The stack contains separately labelled vials, not one combined mixture. A constituent such as GLOW or CJC/ipamorelin is itself a premixed blend, as named in the catalogue.
How is the price calculated?
The selected total is the sum of the current retail price of each constituent vial, multiplied by its displayed quantity. Two vials means two single-vial prices, not a bulk tier. No additional stack discount is applied.
Does adding this change a paid order?
No. The items enter your current demo bag together. Existing bag lines increase by their stated quantities; paid test orders remain unchanged. Checkout is simulated.
What handling information is available?
Format, strength and quantity are shown for each item. Storage, identity, purity and handling require verified batch-specific documentation. This collection supplies no dosing or cycle schedule.
Can component evidence prove the collection works?
No. Study findings retain their individual experimental context. A shared research theme does not establish the effects or safety of a combination.
Selank and Semax bring two complementary research questions into view: GABA-related signalling and neurotrophic pathways involved in learning and memory. A focused starting point for exploring stress-response and cognitive neuroscience.
Explore growth-hormone signalling and cellular adaptation with two vials each of CJC-1295 with DAC, ipamorelin and MOTS-c. This collection brings growth-axis and mitochondrial research into one clearly specified selection.